Researchers have identified a crucial protein, PfSnf2L, that regulates gene expression in the malaria parasite Plasmodium falciparum.

The study, published in Nature, shows that PfSnf2L controls the parasite’s growth and differentiation, essential for its survival and transmission.

The study also found a potential drug, NH125, which blocks this protein’s function, disrupting parasite growth inside red blood cells and preventing the formation of gametocytes, needed for malaria transmission.

This discovery introduces a potential new class of anti-malarial drugs that could help combat the disease by targeting its ability to spread.

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